Biomedical engineers at Boston University and colleagues at the University of Vermont have begun cracking one of the most life-threatening secrets behind the relationship between silent hypoxia and coronavirus following various scenarios.
According to Science Daily, researchers are still unaware of the fact that the reason behind this explains why kovids stop providing oxygen to the patient’s lung bloodstream. All findings will be made with the help of computer models and comparisons with actual patient data.
Silent hypoxia is a condition when the oxygen level in the body is abnormally low, which can damage vital organs of the body for a long time.
Despite experiencing dangerously low levels of oxygen, many people infected with severe cases of Kovid-19 sometimes show no symptoms of shortness of breath or difficulty breathing. The ability to silently damage hypoxia is why it is coined “silence”.
According to research, coronoviruses are believed to first damage the lungs so that they can function properly. Those tissues then lose oxygen, leading to silent hypoxia. But exactly how this happens is still under the table. “We don’t know [how this] It was physically possible, ”says Bella Sookie, one of the study’s authors, when many patients showed almost no signs of abnormalities for a lung scan.
The results of the research, obtained after an in-depth study with the help of a computer model, have been published in Nature Communications which revealed the study of Jacob Herrmann, the lead author of the new study. It added, “Silent hypoxia is possibly caused by a combination of biological mechanisms that can occur simultaneously in the lungs of Kovid-19 patients.”
The good thing the researchers point out is that the lack of sufficient amounts of oxygen due to the infection leads to the ability of the lungs to constrict blood vessels, which then flow through the lung tissue crammed with oxygen throughout the body. Forces to do.
According to Herrmann, “preliminary clinical data suggested that the lungs of some Kovid-19 patients had lost the ability to restrict the flow of blood to previously damaged tissues, and vice versa, possibly causing those blood vessels to become even more Were opening more, which is harder to measure on a CT scan. “
The second scenario observed by researchers with the help of a computer model found that copid-infection can cause silent hypoxia to flare up when the blood vessels lining are inflamed.
The final step was to find out whether Kovid-19 interferes with the normal proportion of blood flow, which is important for the lung to function normally. Researchers found that mismatched air to blood flow ratio is a common symptom in many respiratory diseases such as asthma, and that mismatched lung scans do not appear to be injured or abnormal.
In all, the researchers concluded that a combination of all three factors may contribute to abnormal oxygenation in some Kovid-19 patients, and to achieve perfection in making choices about how patients are treated, clinically, ventilation And it has been suggested to use measures such as supplemental oxygen.
According to the report provided by Science Daily, a number of interventions are currently being studied to allow for a more informed study of the different combination from patient to patient. One of them is a low-tech intervention, with prone positioning that allows patients to blink on their abdomen, pull the back of the lungs into more oxygen and eject mismatched air-to-blood ratios.
(This story is published from a wire agency feed without textual modifications.)
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